How Can Homeopathy Help In The Management Of Thalassemia Patients?
A friend recently asked me whether homeopathy could be used for helping THALASSEMIA patients.
Since it is a purely GENETIC disorder, homeopathy cannot cure thalassemia. Homeopathic drugs cannot repair inherited errors in chromosomes. But homeopathy can reduce the complications of that disease by bringing down the iron over load, without any bad effects even if used for the whole life period. Actually, potentized homeopathic drugs can work as safe chelating agents. Molecular Imprints of IRON contained in potentized forms of various iron-containing drug substances could be used to treat the complications arising from iron overload in thalassemia patients. PULSATILLA 30 if used daily is found to be effective for this purpose. Materia medica of pulsatilla says “it is often indicated after abuse of Iron tonics”, which lead our attention to that drug in “iron over loads”. FERRUM MET 30, FERRUM PHOS 30, FERRUM PICRICUM 30 etc should be used daily for the whole life.
Thalassemia is a form of inherited autosomal recessive blood disorder characterized by abnormal formation of haemoglobin. The abnormal haemoglobin formed results in improper oxygen transport and destruction of red blood cells. Thalassemia is caused by variant or missing genes that affect how the body makes hemoglobin, the protein in red blood cells that carries oxygen. People with thalassemia make less hemoglobin and have fewer circulating red blood cells than normal, which results in mild or severe anemia. Thalassemia will be present as microcytic anemia.
Thalassemia can cause significant complications, including iron overload, bone deformities, and cardiovascular illness.
People with thalassemia can get an overload of iron in their bodies, either from the disease itself or from frequent blood transfusions. Too much iron can result in damage to the heart, liver, and endocrine system, which includes glands that produce hormones that regulate processes throughout the body. The damage is characterized by excessive deposits of iron. Without adequate iron chelation therapy, almost all patients with beta-thalassemia accumulate potentially fatal iron levels.
People with thalassemia have an increased risk of infection. This is especially true if the spleen has been removed.
Thalassemia can make the bone marrow expand, which causes bones to widen. This can result in abnormal bone structure, especially in the face and skull. Bone marrow expansion also makes bones thin and brittle, increasing the risk of broken bones.
The spleen aids in fighting infection and filters unwanted material, such as old or damaged blood cells. Thalassemia is often accompanied by the destruction of a large number of red blood cells and the task of removing these cells causes the spleen to enlarge. Splenomegaly can make anemia worse, and it can reduce the life of transfused red blood cells. Severe enlargement of the spleen may necessitate its removal.
Anemia can cause a child’s growth to slow. Puberty also may be delayed in children with thalassemia.
Diseases, such as congestive heart failure and abnormal heart rhythms, may be associated with severe thalassemia.
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